Autosomal recessive hydrotic ectodermal dysplasia.

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Autosomal recessive hydrotic ectodermal dysplasia.

First cousins, a male and a female, with a new type of hidrotic ectodermal dysplasia are described. They were each the result of first cousin marriage from the Egyptian Karaite community. They both had partial adontia, conical peg-shaped teeth, fine hair that did not grow long, normal sweating, eversion of lips, and pronounced facial similarity. The male had cleft lip on the right side while th...

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Autosomal Recessive Hypohidrotic Ectodermal Dysplasia Caused by a Novel Mutation in EDAR Gene

Backgrounds: Hypohidrotic ectodermal dysplasia (HED) is a rare genetic disorder, distinguished by hypotrichosis, hypohidrosis, and hypodontia. HDE can be inherited in X-linked recessive manner as a result of mutations in the ectodysplasin A (EDA) gene as well as autosomal dominant and autosomal recessive manners both of them caused by mutations in EDA receptor (EDAR) and EDAR-associated death d...

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Hydrotic or Hypohydrotic Ectodermal Dysplasia: Diagnostic Dilemmas (Case Report)

Ectodermal dysplasia includes a large group of syndromes that are clinically and genetically heterogeneous, and are identified by anomalies in structures of ectodermalorigin, and can present with disorders in such structures as hair, nail, teeth, sweat glands, sebaceous glands and conjunctiva and nervous system (Ramesh et al., 2010). This syndrome was first reported by Charles Darwin (1860) (Mi...

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A Novel Splicesite Mutation in the EDAR Gene Causes Severe Autosomal Recessive Hypohydrotic (Anhidrotic) Ectodermal Dysplasia in an Iranian Family

Hypohidrotic ectodermal dysplasia (HED) is a rare congenital disorder arising from deficient development of ectoderm-derived structures including skin, nails, glands and teeth. The phenotype of HED is associated with mutation in EDA, EDAR, EDARADD and NEMO genes, all of them disruptingNF-κB signaling cascade necessary for initiation, formation and differentiation in the embryo and adult. ...

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Mutations in GRHL2 result in an autosomal-recessive ectodermal Dysplasia syndrome.

Grainyhead-like 2, encoded by GRHL2, is a member of a highly conserved family of transcription factors that play essential roles during epithelial development. Haploinsufficiency for GRHL2 has been implicated in autosomal-dominant deafness, but mutations have not yet been associated with any skin pathology. We investigated two unrelated Kuwaiti families in which a total of six individuals have ...

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ژورنال

عنوان ژورنال: Journal of Medical Genetics

سال: 1977

ISSN: 1468-6244

DOI: 10.1136/jmg.14.2.137